All content on this site is intended for healthcare professionals only. By acknowledging this message and accessing the information on this website you are confirming that you are a Healthcare Professional. If you are a patient or carer, please visit Know AML.
Introducing
Now you can personalise
your AML Hub experience!
Bookmark content to read later
Select your specific areas of interest
View content recommended for you
Find out moreThe AML Hub website uses a third-party service provided by Google that dynamically translates web content. Translations are machine generated, so may not be an exact or complete translation, and the AML Hub cannot guarantee the accuracy of translated content. The AML Hub and its employees will not be liable for any direct, indirect, or consequential damages (even if foreseeable) resulting from use of the Google Translate feature. For further support with Google Translate, visit Google Translate Help.
Bookmark this article
During the 61st American Society of Hematology (ASH) meeting in Orlando, US, the AML Global Portal were delighted to speak to Stephen Strickland, Vanderbilt University, Nashville, US. We asked: Why could vosaroxin improve responses to induction chemotherapy in patients with acute myeloid leukemia?
Stephen Strickland explains that vasoroxin has the potential ability to improve upon the response rate of patients with AML, due to the fact is not a P-gp substrate and has p53 independent activity. He also explains that vasoroxin does not cause cardiac toxicity, which means more patients are eligible for its use.
Why could vosaroxin improve responses to induction chemotherapy in patients with AML?
Your opinion matters
Subscribe to get the best content related to AML delivered to your inbox