All content on this site is intended for healthcare professionals only. By acknowledging this message and accessing the information on this website you are confirming that you are a Healthcare Professional. If you are a patient or carer, please visit Know AML.

The AML Hub uses cookies on this website. They help us give you the best online experience. By continuing to use our website without changing your cookie settings, you agree to our use of cookies in accordance with our updated Cookie Policy

Introducing

Now you can personalise
your AML Hub experience!

Bookmark content to read later

Select your specific areas of interest

View content recommended for you

Find out more
  TRANSLATE

The AML Hub website uses a third-party service provided by Google that dynamically translates web content. Translations are machine generated, so may not be an exact or complete translation, and the AML Hub cannot guarantee the accuracy of translated content. The AML Hub and its employees will not be liable for any direct, indirect, or consequential damages (even if foreseeable) resulting from use of the Google Translate feature. For further support with Google Translate, visit Google Translate Help.

Steering CommitteeAbout UsNewsletterContact
LOADING
You're logged in! Click here any time to manage your account or log out.
LOADING
You're logged in! Click here any time to manage your account or log out.
2019-10-31T12:08:30.000Z

ESH 2019 | How do genomic and epigenomic alterations synergize in AML?

Oct 31, 2019
Share:

Bookmark this article

The AML Global Portal were delighted to speak to Lars Bullinger, Charite University Hospital Berlin, Berlin, DE, during the European School of Hematology (ESH) Translational Research Conference on AML. We asked Lars Bullinger: How do genomic and epigenomic alterations synergize in AML?

He explained that epigenetic mutations often predispose to leukemic events. This is why new technologies such as NGS will become part of clinical routine so that we can detect biomarkers  predicting response to more targeted therapies such as hypomethylating agents or FLT3 inhibitors.

How do genomic and epigenomic alterations synergize in AML?

Newsletter

Subscribe to get the best content related to AML delivered to your inbox